mtra2756g.com

MTR A2756G Mutation Basics

MTR A2756G Mutation Basics. MTR A2756G Mutation Basics. The MTR A2756G mutation is a defect of the methionine synthase gene. A mutation of the MTR A2756G gene results in up-regulation of the enzymatic pathway that converts homocysteine into methionine. This enzyme utilizes the methylated form of vitamin B12 to function, and the up-regulation of this enzyme can deplete methyl-B12 levels rapidly. This often results in a deficiency of methyl-B12, which can lead to elevated homocysteine levels. Because vitam.
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The domain mtra2756g.com presently has a traffic classification of zero (the lower the superior). We have traversed one page within the web site mtra2756g.com and found two websites interfacing with mtra2756g.com. We were able to find one contacts and directions for mtra2756g.com to help you communicate with them. The domain mtra2756g.com has been online for six hundred and twenty-five weeks, twenty-three days, fourteen hours, and forty-one minutes.
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MTRA2756G.COM TRAFFIC

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MTRA2756G.COM HISTORY

The domain mtra2756g.com was first submitted to the registrar on February 09, 2013. This website was updated on January 11, 2014. It will go back on the market on the date of February 09, 2015. It is currently six hundred and twenty-five weeks, twenty-three days, fourteen hours, and forty-one minutes young.
REGISTERED
February
2013
UPDATED
January
2014
EXPIRED
February
2015

WEBSITE PERIOD OF EXISTANCE

11
YEARS
11
MONTHS
24
DAYS

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MTRR A66G Mutation Basics

The MTRR A66G mutation is a defect of the methionine synthase reductase gene. A mutation of the MTRR A66G gene results in poor methylation of vitamin B12. If you have high homocysteine, it may be due to the MTRR A66G mutation.

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STEVEN LALEVICH

1254 PIN OAK CT

MARTINSVILLE, IN, 46151

US

1.8122196339
SLALEVIC@INDIANA.EDU

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MTR A2756G Mutation Basics

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MTR A2756G Mutation Basics. MTR A2756G Mutation Basics. The MTR A2756G mutation is a defect of the methionine synthase gene. A mutation of the MTR A2756G gene results in up-regulation of the enzymatic pathway that converts homocysteine into methionine. This enzyme utilizes the methylated form of vitamin B12 to function, and the up-regulation of this enzyme can deplete methyl-B12 levels rapidly. This often results in a deficiency of methyl-B12, which can lead to elevated homocysteine levels. Because vitam.

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The domain states the following, "The MTR A2756G mutation is a defect of the methionine synthase gene." I observed that the web site stated " A mutation of the MTR A2756G gene results in up-regulation of the enzymatic pathway that converts homocysteine into methionine." They also stated " This enzyme utilizes the methylated form of vitamin B12 to function, and the up-regulation of this enzyme can deplete methyl-B12 levels rapidly. This often results in a deficiency of methyl-B12, which can lead to elevated homocysteine levels."

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